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G., “Hypertrophic Scarring and Keloids: Pathomechanisms and Current and Emerging Treatment Strategies,” Molecular Medicine 17 (2011): 113–125
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Some GST classes (especially GST P and GST M) modulate the mitogen-activated protein (MAP) kinase pathway by directly interacting with signaling molecules like c-Jun N-terminal kinase 1 (JNK1) and apoptosis signal-regulating kinase (ASK1), or through post-translational modifications of specific proteins [117,118]
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